D-dimer concentrations and discontinuation of anticoagulants following a thrombotic event
Should the D-dimer concentration be taken into account when considering discontinuation of anticoagulants following a thrombotic event?
Should the D-dimer concentration be taken into account when considering discontinuation of anticoagulants following a thrombotic event?
The role of aspirin in prophylaxis of venous thrombosis is being reevaluated every few years. What is the evidence for the use of aspirin in primary and secondary prophylaxis of venous thrombosis?
Is there any evidence suggesting benefits of D-dimer monitoring in patients with cancer or chronic diseases? As we know that chronic diseases cause elevation in D-dimer levels, should we still measure this parameter to exclude thrombosis?
Are there any important differences in risk stratification, prophylaxis, and treatment of thrombosis in patients with cancer vs patients with nonneoplastic diseases?